Current Issue : January-March Volume : 2026 Issue Number : 1 Articles : 1 Articles
Temporary induced blindness is a reversible loss of visual awareness that occurs despite intact ocular structures and visual pathways. Experimental and clinical evidence indicates that this phenomenon results from dynamic interactions between neural activity, cognitive processes and neurochemical balance within the brain. Neurotransmitters and neuromodulators play a critical role in regulating cortical excitability, perceptual competition and attentional control, all of which influence visual awareness. This review synthesizes current evidence on temporary induced blindness with a specific emphasis on neurochemical mechanisms, including gamma-aminobutyric acid (GABA), glutamate, dopamine, serotonin, acetylcholine and noradrenaline. Experimental paradigms such as motion-induced blindness, visual masking, attentional blink and transcranial magnetic stimulation–induced visual suppression are discussed in relation to excitatory–inhibitory imbalance. Clinical implications are explored in the context of migraine aura, pharmacologically induced visual disturbances and functional visual loss. Understanding the neurochemical modulation of visual awareness provides important insights into visual consciousness and transient perceptual dysfunction....
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